NCL Data2026 · Crystal structure
DataSheet1_Application of novel Fe3O4/Zn-metal organic framework magnetic nanostructures as an antimicrobial agent and magnetic nanocatalyst in the synthesis of heterocyclic compounds.PDF<p>Using the microwave-assisted method, novel Fe<sub>3</sub>O<sub>4</sub>/Zn-metal organic framework magnetic nanostructures were synthesized. The crystallinity, thermal stability, adsorption/desorption isotherms, morphology/size distribution, and magnetic hysteresis of synthesized Fe<sub>3</sub>O<sub>4</sub>/Zn-metal organic framework magnetic nanostructures were characterized by XRD patterns, TG
figshare + Loughborough Research Repository2026 · Astronomical catalogue
Fig 3 E, F and S3 H-L Dataset<p dir="ltr">Dataset underlying Figure 3 E, F as well as S3 H-L - spinal cord myelin sheath lengths and corresponding axon diameters from Piezo1 CKO vs Controls (fl/fl or CNP wt/Cre)</p>
figshare + Loughborough Research Repository2026 · Astronomical catalogue
Fig 3 H and S3 M Dataset<p dir="ltr">Dataset underlying Figure 3H and S3M - % and density of oligodendrocyte lineage cells in the ventral and lateral spinal cord white matter at postnatal day 15</p>
figshare + Loughborough Research Repository2026 · Astronomical catalogue · unknown
Data supporting Compans, Mastrolia et al., 'In vivo modulation of network activity drives the nanoscale reorganisation of axo-axonic synapses at the axon initial segment'. Nature Communications, 2026.<p dir="ltr">See Readme file for details. All files have been scanned for viruses before uploading.</p>
figshare2026 · Astronomical catalogue
Neuronal LAMTOR1 Upregulation Impairs the Autophagy-Lysosomal System in Alzheimer's Disease Brains<p dir="ltr">Dysfunction of the autophagy-lysosomal system is critically involved in the pathogenesis of Alzheimer's diseases (AD). LAMTOR1 is an adaptor protein that resides on late endosomes and lysosomes and participates in the regulation of cellular metabolism and stress responses. This study explore the expression pattern of LAMTOR1 in the AD-affected brains and its roles in AD.</p>