Metal-Induced Localized Microcrystals Buffer Poisoning in NO_x Catalytic Reduction

While traditional acid-base interaction strategies rely on sacrificial sites, such passive defense mechanisms merely mitigate activity loss rather than fundamentally resolving poisoning. This study establishes an active defense mechanism by fabricating amorphous high-entropy metal oxides capable of sequestering metal poisons by inducing localized microcrystallization.

These resulting microcrystals are subsequently transformed into extra active centers for NO_x reduction. Systematic investigation reveals that the K-migration pathway drives a structural evolution from a disordered metastable state to locally ordered nanocrystalline regions, significantly enhancing lattice oxygen mobility. This transition facilitates the activation of NH₃ into O-NH₂^– intermediates.