Pharmacological inhibition of mTORC1 reduces neural death and damage volumen after MCAO by modulating microglial reactivity

Our data show that rapamycin, administered before or after brain ischemia induction, reduced the volume of brain damage and neuronal loss by attenuating the microglial response. Therefore, our findings indicate that the pharmacological inhibition of mTORC1 in the acute phase of ischemia may provide an alternative estrategia to reduce neuronal damage through attenuation of the associated neuroinflammation. This dataset contains the raw data of cuantificación used in this study.

It includes all the observations and measurements that were collected, which are essential for analysis and interpretation. The data is organized in a way that facilitates further exploration and analysis, ensuring that researchers can effectively utilize it for their own studies.